Tags: Gene | Responsible | For | Hearing | Loss | Discovered

Gene Responsible For Hearing Loss Discovered

Friday, 22 June 2007 12:00 AM

According to the BBC, a gene that causes deafness in one in 250 people has been identified by Belgian researchers. Hopefully, it will lead to new treatments for otosclerosis, the most common cause of hearing loss among white males.

Otosclerosis is caused by a malformed bone in the middle ear. The abnormal structure interferes with sound waves passing to the inner ear. Problems usually become noticeable when victims are in their 20s and 30s and realize their hearing is deteriorating. In addition to not being able to hear low-pitched sounds, symptoms include tinnitus, dizziness, or balance problems. Even though the condition can be treated with surgery, hearing loss is often permanent.

By identifying a variant in the gene called TGBF1 that's responsible for otosclerosis, researchers hope they will be able to develop therapies that would either prevent or slow the growth of the abnormal bone that causes the condition.

"The discovery of the gene responsible for a common cause of hearing loss is a fantastic breakthrough for everyone working to improve the lives of deaf and hard of hearing people," said Dr. Catriona Crombie of the Royal National Institute for Deaf People in Great Britain.

© newsmax 2007. All Rights Reserved.

© 2019 Newsmax. All rights reserved.

   
1Like our page
2Share
Pre-2008
According to the BBC, a gene that causes deafness in one in 250 people has been identified by Belgian researchers. Hopefully, it will lead to new treatments for otosclerosis, the most common cause of hearing loss among white males. Otosclerosis is caused by a malformed...
Gene,Responsible,For,Hearing,Loss,Discovered
201
2007-00-22
Friday, 22 June 2007 12:00 AM
Newsmax Media, Inc.
 

Newsmax, Moneynews, Newsmax Health, and Independent. American. are registered trademarks of Newsmax Media, Inc. Newsmax TV, and Newsmax World are trademarks of Newsmax Media, Inc.

NEWSMAX.COM
America's News Page
© Newsmax Media, Inc.
All Rights Reserved